Corrigendum to “Localizing significance of temporal intermittent rhythmic delta activity (TIRDA) in drug-resistant focal epilepsy” [Clin. Neurophysiol. 114 (2003) 70–78]

July 14, 2017 | Autor: Vincenzo Esposito | Categoria: Engineering, Clinical, Drug Resistance, Clinical Neurophysiology
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Clinical Neurophysiology 114 (2003) 954


Corrigendum to “Localizing significance of temporal intermittent rhythmic delta activity (TIRDA) in drug-resistant focal epilepsy” [Clin. Neurophysiol. 114 (2003) 70–78]q Giancarlo Di Gennaroa, Pier Paolo Quaratoa, Paolo Onoratib,c,*, Giovanni B. Colazzaa, Francesco Maria,d, Liliana G. Grammaldoa, Olga Ciccarellie, Nicolo` G. Meldolesia, Fabio Sebastianof, Mario Manfredia,d, Vincenzo Espositoa,g a

Epilepsy Surgery Unit, IRCCS “NEUROMED”, Pozzilli (IS), Italy Child Developmental Center, San Raffaele Pisana-Tosinvest Sanita`, Rome, Italy c Department of Human Physiology and Pharmacology, University “La Sapienza”, Rome, Italy d Department of Neurological Sciences, University “La Sapienza”, Rome, Italy e NMR Research Unit, Institute of Neurology, Queen Square, London, UK f Department of Computer and Systems Science, University “La Sapienza”, Rome, Italy g Department of Neurosurgery, University “La Sapienza”, Rome, Italy b

An error occurred in the 2nd paragraph of the Discussion. The correct paragraph is reproduced below. In order to classify patients with TLE, which represents an heterogeneous clinical group, according to the localization of the epileptogenic zone, we developed a diagnostic grid that is based on correlations among anatomical, electrical and clinical data, in accordance with principles firstly planned by Bancaud et al. (1965) and later developed by Munari et al. (1990), obtained by non-invasive investigations. According to many studies that evaluated the localizing value of a single diagnostic criterion (Williamson et al., 1993; Ebersole and Pacia, 1996; O’Brien et al., 1996; Bleasel et al., 1997; Pacia and Ebersole, 1997; Foldvary et al., 1997, 2001; Mohamed et al., 2001; Henkel et al., 2002; Gil-Nagel and Risinger, 1997; Moriarity et al., 2001), the data obtained from presurgical evaluation were grouped in clusters strongly suggesting the location of epileptogenesis in mesial or lateral aspects of the temporal lobe and, on the basis of our experience, specific modalities of a combination of different

criteria were utilized for localization. With respect to other non-invasive diagnostic protocols (Sperling et al., 1992; Cendes et al., 2000) for TLE surgery, that allow selection of those patients who would benefit from standard temporal lobectomy and identification of those patients who would be invasively investigated or rejected, our diagnostic grid permits the refinement of the classification of TLE in mesial, lateral and mesio-lateral subtypes. The advantage of using this grid is that the surgery can be specifically planned, as far as possible, according to the extension of the epileptogenic zone in the 3 different TLE subtypes (i.e. amygdalohippocampectomy in M-TLE, standard lobectomy in MLTLE, and lesionectomy in lesional L-TLE), avoiding standardized and unnecessary, more extensive surgery. The excellent outcome (. 90% of patients seizure-free), considering both cases with a follow-up of at least 1 year and those with a shorter outcome, showed by the patients who underwent temporal resection, confirms the methodology we used in our study as a reliable diagnostic protocol of TLE.


doi of original article 10.1016/S1388-2457(02)00332-2 Corresponding author. Department of Human Physiology and Pharmacology, University of Rome “La Sapienza”, P.le Aldo Moro 5, 00185 Rome, Italy. Tel.: þ39-06-49910896; fax: þ39-06-49910851. E-mail address: [email protected] (P. Onorati). *

1388-2457/03/$30.00 q 2003 International Federation of Clinical Neurophysiology. Published by Elsevier Science Ireland Ltd. All rights reserved. doi:10.1016/S1388-2475(03)00095-6


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